Open Access
Numéro |
OCL
Volume 18, Numéro 1, Janvier-Février 2011
Dossier : Lipides et inflammation
|
|
---|---|---|
Page(s) | 11 - 13 | |
Section | Nutrition – Santé | |
DOI | https://doi.org/10.1051/ocl.2011.0367 | |
Publié en ligne | 15 janvier 2011 |
- Cani PD, Amar J, Iglesias MA, et al. Metabolic endotoxemia initiates obesity and insulin resistance. Diabetes 2007a ; 56 : 1761–1772. [CrossRef] [PubMed] [Google Scholar]
- Cani PD, Bibiloni R, Knauf C, et al. Changes in gut microbiota control metabolic endotoxemia-induced inflammation in high-fat diet-induced obesity and diabetes in mice. Diabetes 2008 ; 57 : 1470–1481. [CrossRef] [PubMed] [Google Scholar]
- Cani PD, Delzenne NM. Interplay between obesity and associated metabolic disorders : new insights into the gut microbiota. Curr Opin Pharmacol 2009a ; 9 : 737–743. [CrossRef] [Google Scholar]
- Cani PD, Delzenne NM. The role of the gut microbiota in energy metabolism and metabolic disease. Curr Pharm Des 2009b ; 15 : 1546–1558. [CrossRef] [PubMed] [Google Scholar]
- Cani PD, Delzenne NM. The gut microbiome as therapeutic target. Pharmacol Ther 2011. [Google Scholar]
- Cani PD, Neyrinck AM, Fava F, et al. Selective increases of bifidobacteria in gut microflora improve high-fat-diet-induced diabetes in mice through a mechanism associated with endotoxaemia. Diabetologia 2007b ; 50 : 2374–2383. [CrossRef] [PubMed] [Google Scholar]
- Cani PD, Possemiers S, Van de WT, et al. Changes in gut microbiota control inflammation in obese mice through a mechanism involving GLP-2-driven improvement of gut permeability. Gut 2009 ; 58 : 1091–1103. [CrossRef] [PubMed] [Google Scholar]
- Davis JE, Gabler NK, Walker-Daniels J, Spurlock ME. Tlr-4 deficiency selectively protects against obesity induced by diets high in saturated fat. Obesity (Silver Spring) 2008 ; 16 : 1248–1255. [CrossRef] [PubMed] [Google Scholar]
- Erridge C, Samani NJ. Saturated fatty acids do not directly stimulate Toll-like receptor signaling. Arterioscler Thromb Vasc Biol 2009 ; 29 : 1944–1949. [CrossRef] [PubMed] [Google Scholar]
- Erridge C, Webb DJ, Spickett CM. Toll-like receptor 4 signalling is neither sufficient nor required for oxidised phospholipid mediated induction of interleukin-8 expression. Atherosclerosis 2007 ; 193 : 77–85. [CrossRef] [PubMed] [Google Scholar]
- Hotamisligil GS. Inflammation and metabolic disorders. Nature 2006 ; 444 : 860–867. [CrossRef] [PubMed] [Google Scholar]
- Ley RE. Obesity and the human microbiome. Curr Opin Gastroenterol 2010 ; 26 : 5–11. [CrossRef] [PubMed] [Google Scholar]
- Muccioli GG, Naslain D, Backhed F, et al. The endocannabinoid system links gut microbiota to adipogenesis. Mol Syst Biol 2010 ; 6 : 392. [CrossRef] [PubMed] [Google Scholar]
- Rabot S, Membrez M, Bruneau A, et al. Germ-free C57BL/6J mice are resistant to high-fat-diet-induced insulin resistance and have altered cholesterol metabolism. FASEB J 2010 ; 24 : 4948–4959. [PubMed] [Google Scholar]
- Roncon-Albuquerque R Jr., Moreira-Rodrigues M, Faria B, et al. Attenuation of the cardiovascular and metabolic complications of obesity in CD14 knockout mice. Life Sci 2008 ; 83 : 502–510. [CrossRef] [PubMed] [Google Scholar]
- Schwartz EA, Zhang WY, Karnik SK, et al. Nutrient modification of the innate immune response : a novel mechanism by which saturated fatty acids greatly amplify monocyte inflammation. Arterioscler Thromb Vasc Biol 2010 ; 30 : 802–808. [CrossRef] [PubMed] [Google Scholar]
- Shi H, Kokoeva MV, Inouye K, Tzameli I, Yin H, Flier JS. TLR4 links innate immunity and fatty acid-induced insulin resistance. J Clin Invest 2006 ; 116 : 3015–3025. [CrossRef] [PubMed] [Google Scholar]
- Vijay-Kumar M, Aitken JD, Carvalho FA, et al. Metabolic syndrome and altered gut microbiota in mice lacking Toll-like receptor 5. Science 2010 ; 328 : 228–231. [Google Scholar]
Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.
Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.
Le chargement des statistiques peut être long.