Open Access
Numéro
OCL
Volume 23, Numéro 3, May-June 2016
Numéro d'article D306
Nombre de pages 10
Section Dossier: Lipid consumption and functionality: new perspectives / Consommations et fonctionnalités des lipides : nouveaux horizons
DOI https://doi.org/10.1051/ocl/2016009
Publié en ligne 25 mars 2016
  • Amar J, Burcelin R, et al. 2008. Energy intake is associated with endotoxemia in apparently healthy men. Am. J. Clin. Nutr. 87: 1219–1223. [Google Scholar]
  • Bazil V, Strominger JL. 1991. Shedding as a mechanism of down-modulation of CD14 on stimulated human monocytes. J. Immunol. 147: 1567–1574. [PubMed] [Google Scholar]
  • Blackwell TS, Christman JW. 1996. Sepsis and cytokines: current status. British J. Anaesthesia 77: 110–117. [CrossRef] [Google Scholar]
  • Bourlieu C, Michalski MC. 2015. Structure-function relationship of the milk fat globule. Curr. Opin. Clin. Nutr. Metab. Care 18: 118–127. [Google Scholar]
  • Cani PD, Delzenne NM. 2010. Impact du microbiote intestinal sur la perméabilité intestinale et les désordres métaboliques liés à l’obésité. Louvain Med. 129: S8–10. [Google Scholar]
  • Cani PD, Amar J, et al. 2007. Metabolic endotoxemia initiates obesity and insulin resistance. Diabetes 56: 1761–1772. [CrossRef] [PubMed] [Google Scholar]
  • Cani PD, Bibiloni R, et al. 2008. Changes in gut microbiota control metabolic endotoxemia-induced inflammation in high-fat diet-induced obesity and diabetes in mice. Diabetes 57: 1470–1481. [CrossRef] [PubMed] [Google Scholar]
  • Cani PD, Rottier O, et al. 2008. Changes in gut microbiota control intestinal permeability-induced inflammation in obese and diabetic mice through unexpected dependent mechanisms. Diabetologia 51: S34–S35. [Google Scholar]
  • Clemente-Postigo M, Queipo-Ortuno MI, et al. 2012. Endotoxin increase after fat overload is related to postprandial hypertriglyceridemia in morbidly obese patients. J. Lipid Res. 53: 973–978. [Google Scholar]
  • Creely SJ, McTernan PG, et al. 2007. Lipopolysaccharide activates an innate immune system response in human adipose tissue in obesity and type 2 diabetes. Am. J. Physiol. Endocrinol. Metab. 292: E740–E747. [Google Scholar]
  • De Courten B, Moreno-Navarrete JM, et al. 2016. Contrasting association of circulating sCD14 with insulin sensitivity in non-obese and morbidly obese subjects. Mol. Nutr. Food Res. 60: 103–109. [CrossRef] [PubMed] [Google Scholar]
  • De La Serre CB, Ellis CL, et al. 2010. Propensity to high-fat diet-induced obesity in rats is associated with changes in the gut microbiota and gut inflammation. Am. J. Physiol. Gastrointest. Liver Physiol. 299: G440–448. [CrossRef] [PubMed] [Google Scholar]
  • Deopurkar R, Ghanim H, et al. 2010. Differential effects of cream, glucose, and orange juice on inflammation, endotoxin, and the expression of Toll-like receptor-4 and suppressor of cytokine signaling-3. Diabetes Care 33: 991–997. [CrossRef] [PubMed] [Google Scholar]
  • Ding S, Lund PK. 2011. Role of intestinal inflammation as an early event in obesity and insulin resistance. Curr. Opin. Clin. Nutr. Metab. Care 14: 328–333. [CrossRef] [PubMed] [Google Scholar]
  • Eichbaum EB, Harris HW, et al. 1991. Chylomicrons can inhibit endotoxin activity in vitro. J. Surg. Res. 51: 413–416. [Google Scholar]
  • Erridge C, Attina T, et al. 2007. A high-fat meal induces low-grade endotoxemia: evidence of a novel mechanism of postprandial inflammation. Am. J. Clin. Nutr. 86: 1286–1292. [CrossRef] [PubMed] [Google Scholar]
  • Fernandez-Real JM, Perez del Pulgar S, et al. 2011. CD14 modulates inflammation-driven insulin resistance. Diabetes 60: 2179–2186. [CrossRef] [PubMed] [Google Scholar]
  • Frazier TH, DiBaise JK, et al. 2011. Gut microbiota, intestinal permeability, obesity-induced inflammation, and liver injury. J. Parent. Enteral Nutr. 35: 14S–20S. [CrossRef] [Google Scholar]
  • Furet J-P, Kong L-C, et al. 2010. Differential adaptation of human gut microbiota to bariatric surgery-induced weight loss: links with metabolic and low-grade inflammation markers. Diabetes 59: 3049–3057. [CrossRef] [PubMed] [Google Scholar]
  • Gallay P, Barras C, et al. 1994. Lipopolysaccharide (LPS)-binding protein in human serum determines the tumor necrosis factor response of monocytes to LPS. J. Infect. Dis. 170: 1319–1322. [Google Scholar]
  • Gallier S, Vocking K, et al. 2015. A novel infant milk formula concept: Mimicking the human milk fat globule structure. Colloids Surf. B 136: 329–339. [Google Scholar]
  • Gautier T, Klein A, et al. 2008. Effect of plasma phospholipid transfer protein deficiency on lethal endotoxemia in mice. J. Biol. Chem. 283: 18702–18710. [Google Scholar]
  • Ghanim H, Abuaysheh S, et al. 2009. Increase in plasma endotoxin concentrations and the expression of Toll-like receptors and suppressor of cytokine signaling-3 in mononuclear cells after a high-fat, high-carbohydrate meal: implications for insulin resistance. Diabetes Care 32: 2281–2287. [CrossRef] [PubMed] [Google Scholar]
  • Ghanim H, Sia CL, et al. 2010. Orange juice neutralizes the proinflammatory effect of a high-fat, high-carbohydrate meal and prevents endotoxin increase and Toll-like receptor expression. Am. J. Clin. Nutr. 91: 940–949. [CrossRef] [PubMed] [Google Scholar]
  • Ghoshal S, Witta J, et al. 2009. Chylomicrons promote intestinal absorption of lipopolysaccharides. J. Lipid Res. 50: 90–97. [CrossRef] [Google Scholar]
  • Gummesson A, Carlsson LM, et al. 2011. Intestinal permeability is associated with visceral adiposity in healthy women. Obesity (Silver Spring) 19: 2280–2282. [CrossRef] [PubMed] [Google Scholar]
  • Hailman E, Lichenstein HS, et al. 1994. Lipopolysaccharide (LPS)-binding protein accelerates the binding of LPS to CD14. J. Exp. Med. 179: 269–277. [Google Scholar]
  • Harris HW, Grunfeld C, et al. 1993. Chylomicrons alter the fate of endotoxin, decreasing tumor necrosis factor release and preventing death. J. Clin. Invest. 91: 1028–1034. [Google Scholar]
  • Harris HW, Rockey DC, et al. 1998. Chylomicrons alter the hepatic distribution and cellular response to endotoxin in rats. Hepatology 27: 1341–1348. [CrossRef] [PubMed] [Google Scholar]
  • Harte AL, Varma MC, et al. 2012. High fat intake leads to acute postprandial exposure to circulating endotoxin in type 2 diabetic subjects. Diabetes Care 35: 375–382. [CrossRef] [PubMed] [Google Scholar]
  • Hiki N, Berger D, et al. 1999. Changes in endotoxin-binding proteins during major elective surgery: Important role for soluble CD14 in regulation of biological activity of systemic endotoxin. Clin. Diagn. Lab. Immunol. 6: 844–850. [Google Scholar]
  • Kelly CJ, Colgan SP, et al. 2012. Of microbes and meals: the health consequences of dietary endotoxemia. Nutr. Clin. Practice 27: 215–225. [CrossRef] [Google Scholar]
  • Kemp DM, 2013. Does chronic low-grade endotoxemia define susceptibility of obese humans to insulin resistance via dietary effects on gut microbiota? Adipocyte 2: 188–190. [CrossRef] [PubMed] [Google Scholar]
  • Lakatos PL, Kiss LS, et al. 2011. Serum Lipopolysaccharide-binding Protein and Soluble CD14 Are Markers of Disease Activity in Patients with Crohn’s Disease. Inflamm. Bowel Dis. 17: 767–777. [Google Scholar]
  • Lamping N, Dettmer R, et al. 1998. LPS-binding protein protects mice from septic shock caused by LPS or gram-negative bacteria. J. Clin. Invest. 101: 2065–2071. [CrossRef] [PubMed] [Google Scholar]
  • Lassenius MI, Pietilainen KH, et al. 2011. Bacterial endotoxin activity in human serum is associated with dyslipidemia, insulin resistance, obesity, and chronic inflammation. Diabetes Care 34: 1809–1815. [CrossRef] [PubMed] [Google Scholar]
  • Laugerette F, Vors C, et al. 2011. Emulsified lipids increase endotoxemia: possible role in early postprandial low-grade inflammation. J. Nutr. Biochem. 22: 53–59. [Google Scholar]
  • Laugerette F, Furet JP, et al. 2012. Oil composition of high-fat diet affects metabolic inflammation differently in connection with endotoxin receptors in mice. Am. J. Physiol. Endocrinol. Metab. 302: E374–386. [CrossRef] [PubMed] [Google Scholar]
  • Laugerette F, Alligier M, et al. 2014. Overfeeding increases postprandial endotoxemia in men: Inflammatory outcome may depend on LPS transporters LBP and sCD14. Mol. Nutr. Food Res. 58: 1513–1518. [Google Scholar]
  • Laugerette F, Pineau G, et al. 2015. Endotoxemia analysis by the Limulus amoebocyte lysate assay in different mammal species used in metabolic studies J. Anal. Bioanalytical Tech., final revision. [Google Scholar]
  • Le Chatelier ENT, Qin J, Prifti E, et al. 2013. Richness of human gut microbiome correlates with metabolic markers. Nature 29: 541–546. [CrossRef] [PubMed] [Google Scholar]
  • Lecomte M, Bourlieu C, et al. 2015. Milk Polar Lipids Affect In Vitro Digestive Lipolysis and Postprandial Lipid Metabolism in Mice. J. Nutr. 145: 1770–1777. [Google Scholar]
  • Lecomte M, Couedelo L, et al. 2016. Dietary emulsifiers from milk and soybean differently impact adiposity and inflammation in association with modulation of colonic goblet cells in high-fat fed mice. Mol. Nutr. Food Res. DOI:10.1002/mnfr.201500703. [Google Scholar]
  • Ley RE. 2010. Obesity and the human microbiome. Curr. Opin. Gastroenterol. 26: 5–11. [CrossRef] [PubMed] [Google Scholar]
  • Ley RE, Turnbaugh PJ, et al. 2006. Microbial ecology – Human gut microbes associated with obesity. Nature 444: 1022–1023. [CrossRef] [PubMed] [Google Scholar]
  • Libby P. 2002. Inflammation in atherosclerosis. Nature 420: 868–874. [CrossRef] [PubMed] [Google Scholar]
  • Lundman P, Boquist S, et al. 2007. A high-fat meat is accompanied by increased plasma interleukin-6 concentrations. Nutr. Metab. Cardiovasc. Diseas. 17: 195–202. [CrossRef] [Google Scholar]
  • Magné J, Mariotti F, et al. 2010. Early postprandial low-grade inflammation after high-fat meal in healthy rats: possible involvement of visceral adipose tissue. J. Nutr. Biochem. 21: 550–555. [CrossRef] [Google Scholar]
  • Mani V, Hollis JH, et al. 2013. Dietary oil composition differentially modulates intestinal endotoxin transport and postprandial endotoxemia. Nutr. Metab. 10: 6. [CrossRef] [Google Scholar]
  • Marshall JC, 2005. Lipopolysaccharide: an endotoxin or an exogenous hormone? Clin. Infect. Dis. 41: S470–480. [CrossRef] [PubMed] [Google Scholar]
  • Mathison JC, Tobias PS, et al. 1992. Plasma lipopolysaccharide (LPS)-binding protein. A key component in macrophage recognition of gram-negative LPS. J. Immunol. 149: 200–206. [PubMed] [Google Scholar]
  • Matsushita H, Ohta S, et al. 2010. Endotoxin tolerance attenuates airway allergic inflammation in model mice by suppression of the T-cell stimulatory effect of dendritic cells. Int. Immunol. 22: 739–747. [Google Scholar]
  • Michalski MC, Genot C, et al. 2013. Multiscale structures of lipids in foods as parameters affecting fatty acid bioavailability and lipid metabolism. Progress Lipid Res. 52: 354–373. [CrossRef] [Google Scholar]
  • Moreno-Navarrete JM, Escote X, et al. 2013. A role for adipocyte-derived lipopolysaccharide-binding protein in inflammation- and obesity-associated adipose tissue dysfunction. Diabetologia 56: 2524–2537. [CrossRef] [PubMed] [Google Scholar]
  • Nappo F, Esposito K, et al. 2002. Postprandial endothelial activation in healthy subjects and in type 2 diabetic patients: Role of fat and carbohydrate meals. J. Am. College Cardiol. 39: 1145–1150. [CrossRef] [Google Scholar]
  • Neal MD, Leaphart C, et al. 2006. Enterocyte TLR4 mediates phagocytosis and translocation of bacteria across the intestinal barrier. J. Immunol. 176: 3070–3079. [CrossRef] [PubMed] [Google Scholar]
  • Osborn MJ, Rosen SM, et al. 1964. Lipopolysaccharide of the Gram-Negative Cell Wall. Science 145: 783–789. [CrossRef] [PubMed] [Google Scholar]
  • Pais de Barros JP, Gautier T, et al. 2015. Quantitative lipopolysaccharide analysis using HPLC/MS/MS and its combination with the limulus amebocyte lysate assay. J. Lipid Res. 56: 1363–1369. [CrossRef] [PubMed] [Google Scholar]
  • Pastor Rojo O, Lopez San Roman A., et al. 2007. Serum lipopolysaccharide-binding protein in endotoxemic patients with inflammatory bowel disease. Inflamm. Bowel Dis. 13: 269–277. [Google Scholar]
  • Ross R, 1999. Atherosclerosis is an inflammatory disease. Am. Heart J. 138: S419–420. [Google Scholar]
  • Schiffrin EJ, Parlesak A, et al. 2009. Probiotic yogurt in the elderly with intestinal bacterial overgrowth: endotoxaemia and innate immune functions. Br. J. Nutr. 101: 961–966. [CrossRef] [PubMed] [Google Scholar]
  • Sussman M. 2002. Academic: San Diego, CA. [Google Scholar]
  • Tobias PS, Mathison J, et al. 1992. Participation of lipopolysaccharide-binding protein in lipopolysaccharide-dependent macrophage activation. Am. J. Respiratory Cell Mol. Biol. 7: 239–245. [CrossRef] [Google Scholar]
  • Van Dielen FM, van’t Veer C, et al. 2001. Increased leptin concentrations correlate with increased concentrations of inflammatory markers in morbidly obese individuals. Int. J. Obes. Relat. Metab. Disord. 25: 1759–1766. [Google Scholar]
  • Vors C, Gayet-Boyer C, et al. 2014. Produits laitiers et inflammation métabolique : quels liens en phase postprandiale et à long terme? Cah. Nutr. Diet. 50: 25–38. [Google Scholar]
  • Vors C, Pineau G, et al. 2015. Postprandial Endotoxemia Linked With Chylomicrons and Lipopolysaccharides Handling in Obese Versus Lean Men: A Lipid Dose-Effect Trial. J. Clin. Endocrinol. Metab. 100: 3427–3435. [CrossRef] [PubMed] [Google Scholar]
  • Vreugdenhil AC, Rousseau CH, et al. 2003. Lipopolysaccharide (LPS)-binding protein mediates LPS detoxification by chylomicrons. J. Immunol. 170: 1399–1405. [Google Scholar]
  • Wurfel MM, Kunitake ST, et al. 1994. Lipopolysaccharide (LPS)-binding protein is carried on lipoproteins and acts as a cofactor in the neutralization of LPS. J. Exp. Med. 180: 1025–1035. [CrossRef] [PubMed] [Google Scholar]
  • Zhang HS, DiBaise JK, et al. 2009. Human gut microbiota in obesity and after gastric bypass. Proc. Natl. Acad. Sci. USA 106: 2365–2370. [Google Scholar]

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